A cingulate-hippocampal circuit mediates early depressive-like behavior in the mouse model of Alzheimer disease

β₂-Microglobulin coaggregates with Aβ and contributes to amyloid pathology and cognitive deficits in Alzheimer's disease model mice

Minocycline protects against microgliopathy in a <i>Csf1r</i> haplo-insufficient mouse model of adult-onset leukoencephalopathy with axonal spheroids and pigmented glia (ALSP)

b2-microglobulin functions as an endogenous NMDAR antagonist to impair synaptic function

Hypothalamic Menin regulates systemic aging and cognitive decline

Menin Reduces Parvalbumin Expression and is Required for the Anti-Depressant Function of Ketamine

Metabolic reprogramming in astrocytes results in neuronal dysfunction in intellectual disability

A Novel CNTNAP2 Mutation Results in Abnormal Neuronal E/I Balance

Profiling of Sexually Dimorphic Genes in Neural Cells to Identify Eif2s3y, Whose Overexpression Causes Autism-Like Behaviors in Male Mice

SNX14 deficiency-induced defective axonal mitochondrial transport in Purkinje cells underlies cerebellar ataxia and can be reversed by valproate

Trisomy 21-induced dysregulation of microglial homeostasis in Alzheimer's brains is mediated by USP25

Overexpression of Human SNX27 Enhances Learning and Memory Through Modulating Synaptic Plasticity in Mice

TMEM59 Haploinsufficiency Ameliorates the Pathology and Cognitive Impairment in the 5xFAD Mouse Model of Alzheimer's Disease

Cyclin-Dependent Kinase 5-Dependent BAG3 Degradation Modulates Synaptic Protein Turnover

The deubiquitinase USP6 affects memory and synaptic plasticity through modulating NMDA receptor stability

RPS23RG1 Is Required for Synaptic Integrity and Rescues Alzheimer's Disease-Associated Cognitive Deficits

Soluble TREM2 ameliorates pathological phenotypes by modulating microglial functions in an Alzheimer's disease model (vol 10, 1365, 2019)

Soluble TREM2 ameliorates pathological phenotypes by modulating microglial functions in an Alzheimer's disease model

Menin Deficiency Leads to Depressive-like Behaviors in Mice by Modulating Astrocyte-Mediated Neuroinflammation

Neuron-Specific Menin Deletion Leads to Synaptic Dysfunction and Cognitive Impairment by Modulating p35 Expression

Implications of GABAergic Neurotransmission in Alzheimer's Disease