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WOK 论文 10 篇;
Alzheimer's disease: insights into pathology, molecular mechanisms, and therapy
β<sub>2</sub>-Microglobulin coaggregates with Aβ and contributes to amyloid pathology and cognitive deficits in Alzheimer's disease model mice
b2-microglobulin functions as an endogenous NMDAR antagonist to impair synaptic function
Microglial Tmem59 Deficiency Impairs Phagocytosis of Synapse and Leads to Autism-Like Behaviors in Mice
USP25 inhibition ameliorates Alzheimer's pathology through the regulation of APP processing and A beta generation
Metabolic reprogramming in astrocytes results in neuronal dysfunction in intellectual disability
SNX14 deficiency-induced defective axonal mitochondrial transport in Purkinje cells underlies cerebellar ataxia and can be reversed by valproate
Trisomy 21-induced dysregulation of microglial homeostasis in Alzheimer's brains is mediated by USP25
Overexpression of Human SNX27 Enhances Learning and Memory Through Modulating Synaptic Plasticity in Mice
The deubiquitinase USP6 affects memory and synaptic plasticity through modulating NMDA receptor stability